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I agree with your analysis. What I'm wondering about is why it will always be there at it's core since a lot of people do self-cure overtime. If we we look at it from a perspective of neuroplasticity...

You guys who stutter less on caffeine have a lower dopamine baseline. If increasing dopamine makes you more fluent, then you are a subset of people who would benefit from a dopamine agonist medicatio...

Sadly you will need to live the rest lf your life with it, it's just the unfortunate reality to the situation. There's a small chance you'll just grow out of it but the closer you get to your 20's the...

If someone does respond to singing/whispering/background sound improvements to fluency, it usually points towards the basal ganglia, which helps time speech movements and regulate dopamine-based motor...

I'm with you. The basal ganglia neurotransmitter abnormalities and motor desynchronisation are major components of stuttering pathophysiology. Unfortunately it is unknown whether these correlations ar...

That’s right, because neurologically the brain controls all functions of our respiratory and speech systems such as breathing, laryngeal articulation etc…. Too many areas of the brain involved in the ...

Singing uses a different area of the brain to normal speech, and relies on another external timing mechanism as opposed to normal speech, which uses an internal timing mechanism, that’s obviously “fau...

The stuttering is caused by mis-timing / desynchronisation of neurotransmitter signals in the speech motor areas of the brain (basal ganglia, striatum, Broca’s area etc). By slowing down, breathing a...

I wish it was that simple, but there's a lot going on neurologically to result in what we call a block or a repetition which is what you described For instance, the motor systems of the brain not coo...

Neuroplasticy is a beautiful thing. It’s all about rewiring the brain. I wish more PWS could take action like Saji and others and actually work into better themselves because the results are great as ...

In my opinion: I think the model that Sanjeeva visualizes (between speech planning and the outward manifestations) seem to cover a tiny speck regarding the missing gap by Van Riper (and others). As ...

**Part #2:** About distraction: I feel it is not promoted by speech language therapists because, as I say in the abstract, distraction is not reliable. But it helps me even now. I do agree that all...

Stutter theory from Sanjeeva’s (PhD) - Discussions and reflections

Stutter theory from Sanjeeva’s (PhD) - Discussions and reflections Many SLPs and researchers base their work on Charles Van Riper’s theory. Stuttering has clear genetic and neurological roots, and its...

Yes it’s very fascinating! It seems there’s a very fine balance between all neurotransmitters in order for fluent speech to occur. For stutterers, we have dysregulated neurotransmitters in the speec...

that is sosos interesting and i'll try bringing it up because i was never told about dopamine agonist/antagonist !...

Dopamine antagonists can improve fluency significantly if you’re one of those PWS with a hyperactive dopaminergic system. Usually takes a few days to take effect as the brain chemistry adjusts to the...

Try reading aloud to the beat of a Metronome at 80bpm and see if that helps you improve. Do it daily. As you get better, gradually increase the metronome pace to 100bpm, then 120bpm and so forth. It’l...

That’s also a dopamine D2 receptor antagonist, meaning it blocks dopamine activity strongly, even more so than Risperidone does, which is more moderate on D2 receptors. This further reinforces the fa...

Yah this phenomenon is called Choral reading/speaking/singing. It works because it re-synchronizes the brain’s speech timing networks through external rhythm, which can be another voice, or even by u...

Olanzapine is another Dopamine antagonist, which means it essentially blocks dopamine in certain brain regions, and it also reduces seratonins inhibition of dopamine too. Whether it works for you or ...